Everything today appears to be back at the baseline. I will be on Femara for the next 5 days. I’ll go back for a follow-up on the 27th. Normally I’d probably go in sooner, but, um, Christmas is in there and not only are they probably not open, but I will not even be in town.
I actually did get to see the Dr. today. Normally I just see the nurse. I pointed out a bit more family history I recently thought of. He did not seem as alarmed about that family history as some of my others I’ve told him previously. I also asked if it would be ok to take the low-dose aspirin for the entire cycle. He seemed to think it wouldn’t really help, but it could, and if that’s what I wanted to do.. kinda attitude.
So why do I want to try the aspirin? Aspirin therapy can possibly help with the clotting problems, assuming I may have some sort of clotting problem, which we don’t know if I do or not. There are also some other studies that say that the aspirin, working as a blood thinning agent, could cause more blood flow to the reproductive organs, which would overall improve their function. There are other studies that say that it doesn’t do anything to help with reproductive function. So it is all still very blurry right now. But, because I’ve had three very early losses, I wanted to try *something* different than I have been. If it helps, great! If not.. then I don’t know. I’ll cross that bridge when I come to it.
Oh, we talked about the path ahead too. Right now it seems that we’re going to try three more cycles of Femara. After that then we’ll start talking about Femara combined with IUI. At least right now I’ve seen that I can get pregnant on the Femara without IUI, so maybe IUI will be unneccessary. The only way to know though is to see what may come of these cycles before then!
Some of my non-infertile friends may not know what the femara is doing. Often as a treatment for unexplained infertility, a doctor will prescribe a medication that will cause superovulation. In a normal cycle, a woman will have one dominant follicle between her two ovaries. The follicle sort of holds the egg before it is released. In superovulation, several follicles are allowed to grow, allowing for mulitple eggs to be released. This is why the odds of twins is increased on these medications. However, my doctor does not seem worried about me producing twins. For some reason, the femara has a lesser chance of twinning than the other common fertility drug, Clomid. So why does superovulation help in unexplained infertility? Well part of the problem with unexplained is they don’t know what’s causing it, so various treatment options are tried. The idea here is that maybe if we throw more eggs into the equation, there’s higher odds that one will implant and be healthy. I’m sure there are scientific papers behind all of this, but I don’t have any handy.
So, now that I’ve bored everybody to death, I am very excited to be able to do another treatment cycle. These doctor ordered breaks just about kill me!
Again, if anyone wants me to maybe go into more detail about these things, holler at me and I will try to explain if I know. If I don’t know, well then that gives me something to research that I hadn’t thought of! I could probably come up with resources for many of the things as well. Most of it is in my head, but I got all of it either from Dr. Google, or my RE at one point in time or another.
Now that I’ve talked about femara and aspirin in one post… I don’t know what I’ll post about later after this!